Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN
Descrição
IL-33 regulates M1/M2 chemokine expression via mitochondrial redox-related mitophagy in human monocytes - ScienceDirect
Frontiers Reactive Oxygen Species in Macrophages: Sources and Targets
Cells, Free Full-Text
Frontiers Reactive oxygen species formation and its effect on CD4+ T cell-mediated inflammation
Summary of the biology of IL-12 [14]. The main physiological producers
The STAT-ROS cycle extends IFN‑induced cancer cell apoptosis
Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
Doxorubicin-Induced Oxidative Stress and Endothelial Dysfunction in Conduit Arteries Is Prevented by Mitochondrial-Specific Antioxidant Treatment
PDF) Mitochondrial oxidative stress drives IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is controlled by Fas
IL-12 signaling pathway. IL-12 activates the Jak/STAT pathway.
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