Fra-1/AP-1 induces EMT in mammary epithelial cells by modulating Zeb1/2 and TGFβ expression
Descrição
Epithelial–mesenchymal transition: The history, regulatory mechanism, and cancer therapeutic opportunities - Huang - 2022 - MedComm - Wiley Online Library
A compromise between Tgfβ and Egfr signaling programs confers the ability to lead heterogeneous collective invasion
TGFβ and EGF signaling orchestrates the AP-1- and p63 transcriptional regulation of breast cancer invasiveness
Epithelial-mesenchymal plasticity through loss of CTCF motif accessibility and protein expression
The integration of Tgfβ and Egfr signaling programs confers the ability to lead heterogeneous collective invasion
Repression of p63 and induction of EMT by mutant Ras in mammary epithelial cells
An EMT–primary cilium–GLIS2 signaling axis regulates mammogenesis and claudin-low breast tumorigenesis
Oncogenic functions of the EMT-related transcription factor ZEB1 in breast cancer, Journal of Translational Medicine
Fra-1/AP-1 induces EMT in mammary epithelial cells by modulating Zeb1/2 and TGFβ expression
Inter- and intra-tumor heterogeneity in EMT states confers functional diversity in cells that lead collective invasion
IJMS, Free Full-Text
Regulation of epithelial-mesenchymal transition by tumor microenvironmental signals and its implication in cancer therapeutics - ScienceDirect
The role of epithelial–mesenchymal transition-regulating transcription factors in anti-cancer drug resistance
IJMS, Free Full-Text
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